EMF Blocking plus EMF Shielding

No matter if you live in a house or apartment or just need to ensure that your house is free from EMFs, there are a number options to limit your exposure. One of the easiest is to limit your use of electronic devices. You could also consider EMF block paint to prevent EMF radiation from reaching your home. Another way to shield your house from EMF radiation is to use an RF shielding canopy. It is a type made of net that has EMF shielding and is used to block EMFs from entering a room. Another option is to have your home fitted with an enclosure that is conductive. They are referred to as Faraday cages.





A number of studies have proven studies have shown that nonionizing EMF can cause antiproliferative effects on HCC cells. The mechanism of AM RF EMF's anticancer activity in vitro is believed to involve down-regulation of cancer stem cells. This could explain the long-term effects observed in some patients with advanced HCC. However, the mechanism of AM EMF's impact on patients suffering from cancer isn't evident.

Effects from AM electromagnetic fields (RFEM) on HCC tumor growth in vivo was studied in mice. The tumors were split in three different groups. First, the group that was unaffected to RF EMF. Second group members were exposed RF EMF at frequencies similar to the frequency used by humans. Third group members were exposed RF EMF at HCC-specific modulation frequencies. The impact of HCCMF on tumors was evaluated against that of RCF. The results revealed that cancers treated with HCCMF showed significant shrinkage. However, emf blocker treated with RCF didn't show evidence of tumour shrinkage.

The mechanism of tumour-specific AM RF EMF may be due to the fact that tumour cells require Cav3*2 T-type voltage calcium channels to promote proliferation and down-regulation. AM RF EMF's ability to inhibit proliferation in HCC cells is mediated by CACNA1H which is a protein which is responsible for the influx of Ca2+ specific to tumours. The results indicate that CACNA1H could have more broader implications for the treatment and diagnosis of many cancers.

The tumors in those in the group that were unaffected EMF from RF, and fed a normal diet of mice. The tumors in the HCCMF group were treated with Huh7 cells after they were 5 to 7 weeks old. The tumors were removed when they showed excessive burden.

The tumors of the three groups showed distinct growth curves. The HCCMF-treated tumors had a significant reduction in the size of the tumour after eight weeks. However, the tumours that were treated using RCF didn't show reduction in size. emf blockers was significant. The tumors treated with RCF showed necrosis, which is typical in tumors that have been that are exposed to RCF. It is possible that this necrosis is caused by the lack of oxygen in the more invasive cancers.

In conclusion, the findings show that AM RF EMF is a powerful source of anticancer activity in vitro and in the vivo. Numerous studies have demonstrated the fact that AM RF EMF produces measurable reduction in tumours in HCC patients. There is a possibility that AM RF EMF causes these effects because of CACNA1H which is a protein involved in tissue-specific Ca2+ influx. Additionally, block emf may cause a lasting influence on the development of HCC tumors in living tissue.