EMF Blocking and EMF Shielding

No matter if you live in an apartment or a house or just want to keep your home free from EMFs, there are a number options to limit your exposure. One of the easiest is to restrict your use of electronic devices. It is also possible to use EMF block paint to prevent EMF radiation from entering your home. Another way to shield your home from EMF radiations is to install an RF shielding canopy. This is a cloth made of net that has EMF shielding. It is utilized to prevent EMFs from entering rooms. Another alternative is to have your house equipped with an enclosure that is conductive. They are referred to as Faraday cages.

Several studies have shown studies have shown that nonionizing EMF can cause antiproliferative properties in HCC cells. The mechanism behind AM RF EMF's anticancer activity in vitro is thought to be based on the downregulation the cancer stem cell. This could be the reason for the long-term response seen in some patients with advanced HCC. But, the reason for AM RF EMF's effect in patients with cancer is not evident.

The effects from AM RF EMF on HCC tumour growth in vivo was studied in mice. emf blocking were divided into 3 groups. First, the group that was unaffected to RF EMF. Another group of participants was subjected to RF EMF at a frequency that is similar to the one used by humans. In the third, they were subjected to RF EMF with HCC-specific modulation frequencies. The effect of HCCMF on the tumours was evaluated against that of RCF. emf blockers indicated that tumours treated with HCCMF had significant shrinkage. However, tumours treated with RCF did not show any evidence of tumour shrinkage.

The mechanism of tumour-specific AM RF EMF may be based on the fact that tumour cells require Cav3*2 type voltage calcium channels for proliferation and down-regulation. AM RF EMF's ability to inhibit proliferation in HCC cells is controlled through CACNA1H which is a protein which is responsible for the influx of Ca2+ specific to tumours. The findings suggest that CACNA1H could have more broader implications for diagnosis and treatment of various cancers.

The tumours of the control group were not exposed EMF from radiofrequency, and fed a normal mouse diet. block emf in the HCCMF group were injected with Huh7 cells at the time they were 5 to 7 weeks old. The tumours were then euthanized when they showed excessive burden.





The tumours in the three groups also displayed different growth curves. The tumors treated with HCCMF had a significant reduction in tumour size after 8 weeks. However, the tumors treated with RCF didn't show reduction in size. The difference was significant. The tumors treated by RCF were able to show necrosis that is common in tumors that have been that are exposed to RCF. There is a possibility that this necrosis was due to an absence of oxygen in the more invasive cancers.

In conclusion, the findings show an AM-RF EMF is a powerful source of anticancer properties in vitro as well as in the vivo. Numerous studies have demonstrated the fact that AM RF EMF produces measurable shrinkage of tumors within HCC patients. The possibility is that the AM EMF produces these effects because of CACNA1H, a protein that is involved in tissue-specific Ca2+ influx. Furthermore, AM RF EMF may exert a sustained effect on the growth of HCC tumours in the vivo.